<?xml version="1.0" encoding="UTF-8"?><xml><records><record><source-app name="Biblio" version="7.x">Drupal-Biblio</source-app><ref-type>17</ref-type><contributors><authors><author><style face="normal" font="default" size="100%">Vrachatis, Dimitrios A</style></author><author><style face="normal" font="default" size="100%">Papathanasiou, Konstantinos A</style></author><author><style face="normal" font="default" size="100%">Kazantzis, Dimitrios</style></author><author><style face="normal" font="default" size="100%">Sanz-Sánchez, Jorge</style></author><author><style face="normal" font="default" size="100%">Giotaki, Sotiria G</style></author><author><style face="normal" font="default" size="100%">Raisakis, Konstantinos</style></author><author><style face="normal" font="default" size="100%">Kaoukis, Andreas</style></author><author><style face="normal" font="default" size="100%">Kossyvakis, Charalampos</style></author><author><style face="normal" font="default" size="100%">Deftereos, Gerasimos</style></author><author><style face="normal" font="default" size="100%">Reimers, Bernhard</style></author><author><style face="normal" font="default" size="100%">Avramides, Dimitrios</style></author><author><style face="normal" font="default" size="100%">Siasos, Gerasimos</style></author><author><style face="normal" font="default" size="100%">Cleman, Michael</style></author><author><style face="normal" font="default" size="100%">Giannopoulos, George</style></author><author><style face="normal" font="default" size="100%">Lansky, Alexandra</style></author><author><style face="normal" font="default" size="100%">Deftereos, Spyridon</style></author></authors></contributors><titles><title><style face="normal" font="default" size="100%">Inflammatory Biomarkers in Coronary Artery Ectasia: A Systematic Review and Meta-Analysis.</style></title><secondary-title><style face="normal" font="default" size="100%">Diagnostics (Basel)</style></secondary-title><alt-title><style face="normal" font="default" size="100%">Diagnostics (Basel)</style></alt-title></titles><dates><year><style  face="normal" font="default" size="100%">2022</style></year><pub-dates><date><style  face="normal" font="default" size="100%">2022 Apr 19</style></date></pub-dates></dates><volume><style face="normal" font="default" size="100%">12</style></volume><language><style face="normal" font="default" size="100%">eng</style></language><abstract><style face="normal" font="default" size="100%">Isolated coronary artery ectasia (CAE) is a relatively rare clinical entity, the pathogenesis of which is poorly understood. More and more evidence is accumulating to suggest a critical inflammatory component. We aimed to elucidate any association between neutrophil to lymphocyte ratio and coronary artery ectasia. A systematic MEDLINE database, ClinicalTrials.gov, medRxiv, Scopus and Cochrane Library search was conducted: 50 studies were deemed relevant, reporting on difference in NLR levels between CAE patients and controls (primary endpoint) and/or on high-sensitive CRP, IL-6, TNF-a and RDW levels (secondary endpoint), and were included in our final analysis. (PROSPERO registration number: CRD42021224195). All inflammatory biomarkers under investigation were found higher in coronary artery ectasia patients as compared to healthy controls (NLR; SMD = 0.73; 95% CI: 0.27-1.20, hs-CRP; SMD = 0.96; 95% CI: 0.64-1.28, IL-6; SMD = 2.68; 95% CI: 0.95-4.41, TNF-a; SMD = 0.50; 95% CI: 0.24-0.75, RDW; SMD = 0.56; 95% CI: 0.26-0.87). The main limitations inherent in this analysis are small case-control studies of moderate quality and high statistical heterogeneity. Our findings underscore that inflammatory dysregulation is implicated in coronary artery ectasia and merits further investigation.</style></abstract><issue><style face="normal" font="default" size="100%">5</style></issue><custom1><style face="normal" font="default" size="100%">http://www.ncbi.nlm.nih.gov/pubmed/35626182?dopt=Abstract</style></custom1></record></records></xml>