<?xml version="1.0" encoding="UTF-8"?><xml><records><record><source-app name="Biblio" version="7.x">Drupal-Biblio</source-app><ref-type>17</ref-type><contributors><authors><author><style face="normal" font="default" size="100%">Bogdanis, G.C.</style></author><author><style face="normal" font="default" size="100%">Nevill, M.E.</style></author><author><style face="normal" font="default" size="100%">Lakomy, H.K.A.</style></author><author><style face="normal" font="default" size="100%">Boobis, L.H.</style></author></authors></contributors><titles><title><style face="normal" font="default" size="100%">Power output and muscle metabolism during and following recovery from 10 and 20 s of maximal sprint exercise in humans</style></title><secondary-title><style face="normal" font="default" size="100%">Acta Physiologica Scandinavica</style></secondary-title></titles><keywords><keyword><style  face="normal" font="default" size="100%">Aerobic metabolism</style></keyword><keyword><style  face="normal" font="default" size="100%">Anaerobic</style></keyword><keyword><style  face="normal" font="default" size="100%">Fatigue</style></keyword><keyword><style  face="normal" font="default" size="100%">Glycolysis</style></keyword><keyword><style  face="normal" font="default" size="100%">pH</style></keyword><keyword><style  face="normal" font="default" size="100%">Phosphocreatine</style></keyword><keyword><style  face="normal" font="default" size="100%">Recovery</style></keyword><keyword><style  face="normal" font="default" size="100%">Sprinting</style></keyword></keywords><dates><year><style  face="normal" font="default" size="100%">1998</style></year><pub-dates><date><style  face="normal" font="default" size="100%">1998</style></date></pub-dates></dates><urls><web-urls><url><style face="normal" font="default" size="100%">http://www.scopus.com/inward/record.url?eid=2-s2.0-0031866962&amp;partnerID=40&amp;md5=3a3607d03e31fe92a149ced034912c99</style></url></web-urls></urls><volume><style face="normal" font="default" size="100%">163</style></volume><pages><style face="normal" font="default" size="100%">261 - 272</style></pages><language><style face="normal" font="default" size="100%">eng</style></language><abstract><style face="normal" font="default" size="100%">On two separate days eight male subjects performed a 10- or 20-s cycle ergometer sprint (randomized order) followed, after 2 min of recovery, by a 30-s sprint. Muscle biopsies were obtained from the vastus lateralis at rest, immediately after the first sprint and after the 2 min of recovery on both occasions. The anaerobic ATP turnover during the initial 10 s of sprint 1 was 129 ± 12 mmol kg dry weight-1 and decreased to 63 ± 10 mmol kg dry weight-1 between the 10th and 20th s of sprint 1. This was a result of a 300% decrease in the rate of phosphocreatine breakdown and a 35% decrease in the glycolytic rate. Despite this 51% reduction in anaerobic ATP turnover, the mean power between 10 and 20 s of sprint 1 was reduced by only 28%. During the same period, oxygen uptake increased from 1.30 ± 0.15 to 2.40 ± 0.23 L min-1, which partially compensated for the decreased anaerobic metabolism. Muscle pH decreased from 7.06 ± 0.02 at rest to 6.94 ± 0.02 after 10 s and 6.82 ± 0.03 after 20 s of sprinting (for all changes P &amp;lt; 0.01). Muscle pH did not change following a 2-min recovery period after both the 10- and 20-s sprints, but phosphocreatine was resynthesized to 86 ± 3 and 76 ± 3% of the resting value, respectively (n.s. 10- vs. 20-s sprint). Following 2 min of recovery after the 10-s sprint subjects were able to reproduce peak but not mean power. Restoration of both mean and peak power following the 20-s sprint was 88% of sprint 1, and was lower compared with that after the 10-s sprint (P &amp;lt; 0.01). Total work during the second 30-s sprint after the 10- and the 20-s sprint was 19.3 ± 0.6 and 17.8 ± 0.5 kJ, respectively (P &amp;lt; 0.01). As oxygen uptake was the same during the 30-s sprints (2.95 ± 0.15 and 3.02 ± 0.16 L min-1), and [Phosphocreatine] before the sprint was similar, the lower work may be related to a reduced glycolytic ATP regeneration as a result of the higher muscle acidosis.</style></abstract><issue><style face="normal" font="default" size="100%">3</style></issue><notes><style face="normal" font="default" size="100%">Cited By (since 1996):100Export Date: 9 November 2014</style></notes></record></records></xml>