@article {7645,
	title = {VWF, WM, and angiogenesis: Is there a link?},
	journal = {Blood},
	volume = {120},
	year = {2012},
	note = {Cited By :2Export Date: 21 February 2017},
	month = {2012},
	pages = {3163 - 3164},
	abstract = {The serum levels of several angiogenic cytokines are increased in patients withWaldenstr{\"o}m macroglobulinemia (WM). Angiopoietin-2 is also increased and the balance of angiopoietin-1/angiopoietin-2, which antagonize for the receptor tyrosine kinase Tie-2 is in favor of angiopoietin-2. Lymphoplasmacytic cells may produce VEGF, but they also produce large amounts of CCL-3 which attracts macrophages, and probably mast cells. Mast cells support lymphoplasmacytic cells and may also produce VEGF. VWF is stored in the Weibel-Palade bodies together with several other mediators, including angiopoietin-2. The endothelial cells (ECs) release the content of Weibel-Palade bodies in response to stimuli, such as VEGF and VWF and other molecules (such as angiopoitein-2) are released simultaneously. VWF may also have an inhibitory activity in the constitutive VEGFR-2-dependent pathway(s), which promote EC migration.},
	url = {https://www.scopus.com/inward/record.uri?eid=2-s2.0-84868149992\&doi=10.1182\%2fblood-2012-08-451468\&partnerID=40\&md5=015b8b19e4740049be1dfb265adf7399},
	author = {Kastritis, E. and Dimopoulos, M.A.}
}
