<?xml version="1.0" encoding="UTF-8"?><xml><records><record><source-app name="Biblio" version="7.x">Drupal-Biblio</source-app><ref-type>17</ref-type><contributors><authors><author><style face="normal" font="default" size="100%">Papaioannou, T.G.</style></author><author><style face="normal" font="default" size="100%">Protogerou, A.D.</style></author><author><style face="normal" font="default" size="100%">Argyris, A.</style></author><author><style face="normal" font="default" size="100%">Aissopou, E.</style></author><author><style face="normal" font="default" size="100%">Georgiopoulos, G.</style></author><author><style face="normal" font="default" size="100%">Nasothimiou, E.</style></author><author><style face="normal" font="default" size="100%">Tountas, C.</style></author><author><style face="normal" font="default" size="100%">Sfikakis, P.P.</style></author><author><style face="normal" font="default" size="100%">Stergiopulos, N.</style></author><author><style face="normal" font="default" size="100%">Tousoulis, D.</style></author></authors></contributors><titles><title><style face="normal" font="default" size="100%">Total arterial compliance, estimated by a novel method, is better related to left ventricular mass compared to aortic pulse wave velocity: The SAFAR study</style></title><secondary-title><style face="normal" font="default" size="100%">Clin Exp Hypertens</style></secondary-title><alt-title><style face="normal" font="default" size="100%">Clinical and experimental hypertension (New York, N.Y. : 1993)</style></alt-title><short-title><style face="normal" font="default" size="100%">Clinical and experimental hypertension (New York, N.Y. : 1993)Clinical and experimental hypertension (New York, N.Y. : 1993)</style></short-title></titles><dates><year><style  face="normal" font="default" size="100%">2017</style></year></dates><number><style face="normal" font="default" size="100%">3</style></number><edition><style face="normal" font="default" size="100%">2017/04/28</style></edition><volume><style face="normal" font="default" size="100%">39</style></volume><pages><style face="normal" font="default" size="100%">271-276</style></pages><isbn><style face="normal" font="default" size="100%">1525-6006 (Electronic)1064-1963 (Linking)</style></isbn><language><style face="normal" font="default" size="100%">eng</style></language><abstract><style face="normal" font="default" size="100%">AIM: The investigation of the association between total arterial compliance (CT)-estimated by a novel technique-with left ventricular mass (LVM) and hypertrophy (LVH). Our hypothesis was that CT may be better related to LVM compared to the gold-standard regional aortic stiffness. Within the frame of the ongoing cross-sectional study &quot;SAFAR,&quot; 226 subjects with established hypertension or with suspected hypertension underwent blood pressure (BP) assessment, carotid-to-femoral pulse wave velocity (cf-PWV), and echocardiographic measurement of LVM. LVM index (LVMI) was calculated by the ratio of LVM to body surface area. CT was estimated by a previously proposed and validated formula: CT = 36.7 /cf-PWV2 [ml/mmHg]. LVMI was related to age (r = 0.207, p = 0.002), systolic BP (r = 0.248, p &amp;lt; 0.001), diastolic BP (r = 0.139, p = 0.04), mean BP (r = 0.212, p = 0.002), pulse pressure (r = 0.212, p = 0.002), heart rate (r = -0.172, p = 0.011), cf-PWV (r = 0.268, p &amp;lt; 0.001), and CT (r = -0.317, p &amp;lt; 0.001). The highest correlation was observed for CT that was significantly stronger than the respective correlation of cf-PWV (p &amp;lt; 0.001). In multivariate analysis, CT was a stronger determinant, compared to cf-PWV, of LVMI and LVH. It remains to be further explored whether CT has also a superior prognostic value beyond and above local or regional (segmental) estimates of pulse wave velocity.</style></abstract><issue><style face="normal" font="default" size="100%">3</style></issue><accession-num><style face="normal" font="default" size="100%">28448191</style></accession-num><notes><style face="normal" font="default" size="100%">Papaioannou, Theodore GProtogerou, Athanase DArgyris, AntonisAissopou, EvangeliaGeorgiopoulos, GeorgeNasothimiou, EfthimiaTountas, ChristosSfikakis, Petros PStergiopulos, NikolaosTousoulis, DimitriosEnglandClin Exp Hypertens. 2017;39(3):271-276. doi: 10.1080/10641963.2016.1247165.</style></notes><auth-address><style face="normal" font="default" size="100%">a Biomedical Engineering Unit, 1st Department of Cardiology , &quot;Hippokration&quot; Hospital, Medical School, National and Kapodistrian University of Athens , Athens , Greece.b Cardiovascular Prevention &amp; Research Unit, Department of Pathophysiology, Medical School , National and Kapodistrian University of Athens , Athens , Greece.c Hypertension Center and Cardiovascular Research Laboratory, 1st Department of Propaedeutic Medicine , &quot;Laiko&quot; Hospital, Medical School, National and Kapodistrian University of Athens , Athens , Greece.d Laboratory of Hemodynamics and Cardiovascular Technology, Institute of Bioengineering , Ecole Polytechnique Federale de Lausanne , Lausanne , Switzerland.</style></auth-address><remote-database-provider><style face="normal" font="default" size="100%">NLM</style></remote-database-provider></record></records></xml>